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Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

A single amino acid change in the vitamin D receptor can disrupt its function and lead to hair loss.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

P-cadherin is crucial for hair follicle pigmentation but not skin pigmentation.

miR-3606-3p reduces skin fibrosis by blocking key signaling pathways.

P-cadherin is important for hair growth and health, and its problems can cause hair and skin disorders.

Fipronil effectively treats Lynxacarus radovskyi in outdoor cats.

P-selectin is not the only factor that prevents scarring in fetal wound healing in mice.

Mummy hair from the Taklamakan desert has calcium and phosphorus inside.

A genetic marker linked to a type of hair loss was found in most patients studied.

Everyone has an immune response to PPD, but reactions differ, causing tolerance, mild inflammation, or allergy.

The p.P25L mutation in the KRT5 gene causes a rare skin condition that worsens over time and may lead to hair loss starting in young adulthood.

Supplementing vitamins and minerals prevented hair loss in Icelandic horses.

The LMNA mutation affects skin structure even in asymptomatic carriers.

A dog with a hereditary skin condition causing blisters and hair loss survived for a year.

A new wound dressing with p-Coumaric acid helps heal diabetic wounds faster by reducing inflammation and promoting skin repair.

VB1953 gel significantly reduced acne and resistant bacteria in patients who didn't respond to clindamycin.

Finasteride increases P-glycoprotein activity in the liver and kidney.

Certain genes may promote longer root hairs in plants when phosphorus is low.

Punica granatum leaf extract may help treat skin fibrosis by reducing inflammation and oxidative stress.

Hair dye ingredient PPD can cause severe allergic reactions, including facial swelling.

ARHGEF3 is essential for proper hair follicle development in mice.